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Ecg Basics and Abnormal Ecg

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ELECTROCARDIO-graphy (E.C.g.)

Uses: - To detect arrhythmias To diagnose chamber hypertrophy To diagnose myocardial infarction
Electrocardiogram is the graphic representation of electrical changes occurring in the cardiac muscle. It is recorded by using surface electrodes. These are of two types:
Bipolar leads
Unipolar leads
Bipolar leads Lead I-Right wrist and left wrist Lead II –Right wrist and left ankle Lead III- Left wrist and left ankle
Unipolar leads - Limb leads and chest leads
a. Limb leads- aVR – right wrist aVL –left wrist aVF – left ankle
b. Chest leads- V1-4th right intercostal space at the sternal border V2-4th left intercostal space at the sternal border V3- between V2 & V4 V4- at the midclavicular line in the left 5th intercostal space V5- at the anterior axillary line in the same horizontal plane V6- at the mid-axillary line in the same horizontal plane
V3R - on the right side – position same as that of V3
V4R - on the right side – position same as that of V4
E.C.g. - - paper speed-25mm/sec horizontally 1mm(one small square)=0.04sec (one large square=0.04x5=0.2sec vertically 1mm=0.1mv
P-wave- atrial depolarisation –upright width – 2.5mm=0.1sec
QRS - ventricular de-polarisation width – 1-2.5mm=0.04-0.1sec average 0.08sec height – variable
PR- interval-from the starting of atrial depolarisation to ventricular depolarization 3-5mm(0.12-0.2sec)
T-wave-upright-repolarisation – widest wave 6.2-7.5mm(0.25-0.3sec) height-1/3 of QRS complex
QT- interval-ventricular depolarisation & repolarisation 8.7mm-10.5mm(0.35-0.42sec)
Differences in shapes of waves in different leads All waves are inverted in aVR
V1-V6— height of R wave –goes on increasing- from V1 to V6 while depth of S wave –goes on decreasing from V1 to V6 .
Myocardial infarction Area of ischaemia – ST depression and T wave inversion or T wave inversion alone Area of injury – ST elevation. Area of infaraction—Presence of Q wave.
Anterior wall MI- Leads Antero septal – V1 to V4 Antero lateral—V4 to V6, Lead 1 and aVL
Inferior wall MI- Leads II, III, aVF

Hyperkalemia - Tall T wave
Hypokalemia - Presence of U wave after T wave.
E.C.g. -- Reading - 7 steps 1 Calculate heart rate- number of QRS complexes in one minute i.e. - ___________1500______________________ no. of small squares in between two R waves.

or ____________300_________________________ no. of large squares in between two R waves. if rhythm is irregular . count the QRS complexes in a 6 second strip and multiply that with 10.

2. Check the P-P interval and RR interval regularity. 3. Examine P wave-presence, position with respect to QRS, and shape 4. Measure PR interval 5. QRS complex – width, shape 6. Examine ST – segment-ST elevation or ST depression 7. T wave-height- ( 1 rd of QRS ) upright 3 -look for T wave inversion.

ARRHYTHMIAS
Conduction- system-rate -- SA node-60-90/mt AV node-40-60/mt

Purkinje fibres – 20-40/mt(idioventricular rhythm)

NON CARDIAC • Hypoxia • Acidosis • Hypercapnoea • Electrolyte imbalances • Hypotension • Coffee,tea,alcohol, smoking • Emotional stress • Drugs

Classification of Arrhythmias

1. Disorders of Impulse Production 2. Disorders of Impulse Conduction

Disorders of Impulse Production 1. SupraVentricular 2. Ventricular

SupraVentricular Arrhythmias

1 Sins bradycardia - rate below 60/mt Causes..Athelets, Hypothyroidism, Inferior wall MI Treatment..Treat the cause, Atropine. 2 Sins tachycardia - rate 100-160/mt Causes..Exercise, Emotion, Hyperthyroidism, Fever, MI, C.C.F. Treatment..Treat the cause, no need of drugs 3. Sick Sinus Syndrome Sometimes tachycardia and sometimes bradycardia and asystole Treatment..Pacemaker 4 Atrial premature contraction-P.A.C. Originates from an ectopic focus and occur before due time. QRS Complexes are of normal shape, but P maybe of abnormal shape 5. Supraventricular tachycardia – SVT Paroxysmal atrial tachycardia-PAT- rate 180 –250/mt Orginates from an ectopic focus, sudden onset and sudden cessation. C/F- Palpitation, sometimes associated with BP fall. Treatment..Vagal stimulation-carotid massage , forcing to vomit drinking coldwater,valsalvamanoeuvre,ivadenosine,verapamil HCL, digoxin, propranolol, if not reverted , DC shock.

position of chest leads & limb leads

[pic][pic]

normal E.C.G
[pic]

[pic]

E.C.G changes in MI

[pic]

Hyperkalemia
[pic]

normal sinus rythm
[pic]

• Sinus Bradycardia
[pic]

sinus tachycardia
[pic]

SVT
[pic]

Atrial Flutter
[pic]

Atrial Fibrillation
[pic]

Junctional Rhythm
[pic]

Accelerated Junctional Rhythm
[pic]

Premature Ventricular Contraction
[pic]

• multifocal PVC's
[pic]

Ventricular bigeminy
[pic]

Ventricular Trigeminy
[pic]

couplets…
[pic][pic]

Triplets
[pic]

[pic]

(VT)
[pic]

VF
[pic]

Asystole -
[pic]

1st Degree AV Block
[pic]

2nd Degree Block Type 1 (Wenckebach)
[pic]

2nd Degree Block Type 2
[pic]
[pic]

3rd Degree Block
[pic]

Bundle Branch Block
[pic]
----------------------- ARRHYTHMIAS - CAUSES

CARDIAC 1. IHD 2. RHD 3. Congenital heart diseases 4. Cardiomyopathy 5. Myocarditis

P 2.5mm = 0.1sec QRS 1- 2.5mm = 0.04 – 0.1sec (0.08sec) PR-Interval 3 – 5mm = 0.12 – 0.2sec, T – 6.2 – 7.5mm = 0.25-0.3sec QT-Interval- 8.7-10.5mm = 0.35-0.42sec

Sinus Tachycardia

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