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Tracheostomy Suctioning

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HUMIDIFICATION
Claudine Billau
The formation of a tracheostomy significantly alters the patient’s respiratory physiology. In bypassing the upper respiratory tract, the patient is more susceptible to changes in humidity and there is a consequential change in the function of the respiratory mucosa. Understanding these changes is fundamental to managing these patients effectively (see Chapter 1, Anatomy and
Physiology of the Respiratory Tract).
NORMAL MECHANISM OF HUMIDIFICATION
The upper respiratory system: the nose, pharynx, larynx and the trachea
(Fig. 1), normally provides an effective system for conditioning inspired gases. As well as acting as a filter for foreign particles and microbes, the upper airway also warms and humidifies inspired gases so that the gas travelling beyond the carina enters the lower airways and the alveoli at body temperature and fully saturated with water vapour.1
As inspired air enters the upper airway and passes over the nasal turbinates and conchae, gas flow becomes turbulent. This leads to an increase in the number of gas molecules coming into contact with the nasal mucosa. The nasal mucosa is highly vascular and is kept moist by a combination of secretions from mucous glands and direct transudation of fluid through cell walls.1,2 The secreted mucus is hydroscopic and its viscosity varies depending on its glycoprotein content.2,3 The turbulent gas flow results in an increasing efficiency in the warming and conditioning of inspired gases by turbulent convection. As the air is warmed, water from the mucosa evaporates and is transferred to the incoming gas.
In normal conditions, when the upper respiratory tract is normal, room air is inhaled at a temperature of around 20C with a relative humidity of 50%.
As it passes across the warmer and more humid mucosa the air becomes progressively warmer and more saturated. When

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