...Fibromyalgia Jamie A Cothran Mod 04 writing assignement Fibromyalgia: The Pathophysiology Abstract The mystery surrounding fibromyalgia has been long and tedious. At first neurology did not want to claim it saying it was a psychiatric problem, then psychiatric said no the pain of the disease gives you depression and anxiety , and they pushed it off to musculoskeletal system. Where ever it starts, for the patient, the pain is real, it’s their perception. It’s not up to us to judge, but up to us to help ease the pain, and hopefully one day find a cure. But first we must understand how it manifests. With no concrete scientific evidence and only theories to date, we can at least see ways to help ease the pain; therapies to help make the patients more active and have a better quality of life. Fibromyalgia: The Pathophysiology Much debate of Fibromyalgia has existed since the first diagnosis. Some people are lead to believe that it replaced Chronic Pain Syndrome and/or Chronic Fatigue Syndrome. Did it, we will examine that here. Depending on who you talk to Fibromyalgia exists, Endocrinology says no and Neurology says yes. Who’s right, which is what we will find out? What we do know; “Fibromyalgia is a chronic musculoskeletal syndrome characterized by widespread joint and muscle pain, fatigue, and tender points” (McCance RN & Huether RN, 2010, p. 1606). If that is Fibromyalgia does that not describe Chronic Pain Syndrome? Well yes and no, Chronic...
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...Therapeutic Exercise Prescription Concepts Applied to Patients Diagnosed With Fibromyalgia Pain is defined as an “unpleasant feeling, conveyed to the brain by sensory neurons” (Medical Dictionary, 2013). Pain is a subjective interpretation, which describes location, intensity and nature of sensory information resulted from noxious stimulation of nerve endings. The emotional response and other various conscious or unconscious responses add to the concept of pain. Acute pain usually results from tissue damage and it usually ends once the injury is healed. Chronic pain is a long term condition related to a persistent or degenerative disease or it doesn’t have an identifiable cause. Although there are different opinions about when a patient can be declared as suffering from chronic pain (after six months or twelve months), the presence of such sensation after the healing period usually leads to this diagnosis. One of the diseases with a generalized chronic pain as a symptom, which continues to generate many debates in the medical world, is fibromyalgia. Fibromyalgia, a disorder that affects the overall system and has an effect of generalized chronic pain, will be delineated in this paper, including its causes, manifestation, and part of the process of managing this disease in which a kinesiologist would have a central role. Fibromyalgia, estimated to affect 4% of the population, is a compilation of symptoms accompanying pain, which include fatigue, stiffness and sleep disturbance...
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...A Caucasian man with Hip Pain The management of pain can be tricky as sometimes-healthcare professions struggle between objective pain and associating the subjective pain which is what the patient says, “It is.” Understanding the Pathophysiology of pain and the cause of acute pain that lead to chronic pain is an essential aspect of managing pain. The nociceptive pain pathway is series of neurons that detect noxious stimuli that end up with pain; however, the pathway starts at the peripheral to the spinal cord and the brain (Stahl 2013). Knowing the pain pathway will help the PMHNP to understand the use of certain psychological drugs for the manage pain like Selective norepinephrine reuptake inhibitors (SNRI) for depression (Mehalick et al. 2016). According to the case study, the patient suffered from right hip pain that has been going...
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...Disease Achondroplasia is the most frequent form of short-limb dwarfism (Nahar R et al 2009). As well as short stature due to shortening of limbs, affected individuals have characteristic facies with frontal bossing and mid-face hypoplasia, exaggerated lumbar lordosis, limitation of elbow extension, genu varum and trident-like hands. Incidence increases with paternal age. Achondroplasia is caused, in virtually all of the cases, by a G380R mutation in fibroblast growth factor receptor 3 (FGFR3) (Di Rocco F et al 2014). FGFR3 is also important in craniofacial, vertebral and neurological development such that this mutation has multiple effects in an affected individual (Horton WA et al 2007). Clinical Presentation Gross motor development frequently is delayed. Motor milestones such as head control and independent sitting, standing, and ambulation may lag by 3-6 months. Speech and language problems may be caused by tongue thrust (due to abnormal maxillomandibular relationship) but often resolve spontaneously. Twenty percent of patients experience delayed speech acquisition. Cognitive skills are preserved, and the intelligence level is within normal limits. Cranial enlargement and poor head control place the infant at risk for extension injuries. An Australian study assessed the functional milestones of achondroplasia children aged 3-7 years. The data noted that while milestones were delayed across all ages studied, functioning improved between age 3 and 5 years, although not...
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...Abstract Since joint destruction begins within a few weeks of symptom onset in rheumatoid arthritis (RA), early diagnosis and treatment are crucial to decrease impairment of physical function and halt disease progression. A delay in therapy of as little as 3 months has demonstrated to cause irreversible joint damage. Treatment plans for RA includes pharmacological as well as non-pharmacological modalities. Treatments for rheumatoid arthritis (RA) have involved a variety of single agent and combination therapies with a goal, to slow disease progression and bone destruction. The research indicates that not all drug combinations are equally effective in all patients with RA. To more accurately distinguish how to best manage early RA, and because treating RA within 3 months of diagnosis appears crucial for improved outcomes, this paper review studies that compare combination to mono-therapies in RA. Introduction Rheumatoid Arthritis (RA) is considered to be an inflammatory arthritis (Gardner, 2010). It is a disease that is chronic and characterized by diffuse inflammation and degeneration of the connective tissues, often characterized by joint stiffness in the morning that lasts at least 30 minutes but can range to several hours (Gardner, 2010). Pain improves with activity as fluid accumulated in the joints during inactivity is forced back into the system through the lymphatics. RA has a different cause from Osteoarthritis (OA). It is considered to be...
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...PATHO Exam 3 Study Guide * Define KEY WORDS (terminology) listed in the syllabus * Answer the OUTCOMES in the syllabus as if they are questions * Review all Activities, Games, extra videos, journal articles, etc. posted in course contents * Review the handouts from class: case studies, matching, charts, etc. Normal Values | Intracranial pressure | 5-10 mm Hg | Blood glucose | 70-130 | Hgb A1c | <5.7% | Thyroid levels | | Parathyroid levels | | Types of bone cells | Osteoblasts | Bone forming cellsThey are responsible for bone growth and repair | Osteocytes | Osteoblasts that have become trapped, imprisoned within mineralized bone matrix (MATURE BONE CELLS) | Osteoclasts | Reabsorb or remove bone during growth and repair (also assist in the release of calcium and phosphate)**bone reabsorption; bone destroying cells | *So, if one is immobilized then the osteoclastic activity is greater than the osteoblastic activity in bone marrow decreases. This is why we have debone mineralization during immobilization. Maintenance of bone integrity | This occurs through remodeling and it is a 3 phase process where existing bone is resorbed and new bone is laid down [repairs bone, does not heal bones] | Phase 1 | Activation phaseThis is where a stimulus occurs, such as a weight baring exercise, causing the formation of osteoclasts | Phase 2 | Resorption This is where osteoclasts form a cutting zone and resorb or remove bone | Phase 3 | Formation...
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...------------------------------------------------- Somatoform disorder From Wikipedia, the free encyclopedia Somatoform disorder | Classification and external resources | ICD-10 | F45 | ICD-9 | 300.8 | DiseasesDB | 1645 | eMedicine | med/3527 | MeSH | D013001 | In psychology, a somatoform disorder is a mental disorder characterized by physical symptoms that suggest physical illness or injury – symptoms that cannot be explained fully by a general medical condition, direct effect of a substance, or attributable to another mental disorder (e.g. panic disorder).[1] The symptoms that result from a somatoform disorder are due to mental factors. In people who have a somatoform disorder, medical test results are either normal or do not explain the person's symptoms. Patients with this disorder often become worried about their health because the doctors are unable to find a cause for their health problems. This causes severe stress, due to preoccupations with the disorder that portrays an exaggerated belief about the severity of the disorder. [2]Symptoms are sometimes similar to those of other illnesses and may last for several years. Usually, the symptoms begin appearing during adolescence, and patients are diagnosed before the age of 25 years. [3] Somatoform disorders are not the result of conscious malingering (fabricating or exaggerating symptoms for secondary motives) or factitious disorders (deliberately producing, feigning, or exaggerating symptoms) – sufferers...
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...PN MENTAL HEALTH NURSING EDITION . CO NT ASTERY SERI ES TM N E R EV MOD IE W LE U PN Mental Health Nursing Review Module Edition 9.0 CONtriButOrs Sheryl Sommer, PhD, RN, CNE VP Nursing Education & Strategy Janean Johnson, MSN, RN Nursing Education Strategist Sherry L. Roper, PhD, RN Nursing Education Strategist Karin Roberts, PhD, MSN, RN, CNE Nursing Education Coordinator Mendy G. McMichael, DNP, RN Nursing Education Specialist and Content Project Coordinator Marsha S. Barlow, MSN, RN Nursing Education Specialist Norma Jean Henry, MSN/Ed, RN Nursing Education Specialist eDitOrial aND PuBlisHiNg Derek Prater Spring Lenox Michelle Renner Mandy Tallmadge Kelly Von Lunen CONsultaNts Deb Johnson-Schuh, RN, MSN, CNE Loraine White, RN, BSN, MA PN MeNtal HealtH NursiNg i PN MeNtal HealtH NursiNg review Module editioN 9.0 intellectual Property Notice ATI Nursing is a division of Assessment Technologies Institute®, LLC Copyright © 2014 Assessment Technologies Institute, LLC. All rights reserved. The reproduction of this work in any electronic, mechanical or other means, now known or hereafter invented, is forbidden without the written permission of Assessment Technologies Institute, LLC. All of the content in this publication, including, for example, the cover, all of the page headers, images, illustrations, graphics, and text, are subject to trademark, service mark, trade dress, copyright, and/or other intellectual property rights or licenses...
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