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Case Study About Acute Glomerulonephritis

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Submitted By shielavaldez
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Nueva Ecija Doctors’ Colleges, Inc.
Cagayan Valley Road, Cabanatuan City
College of Medical Technology

Acute Glomerulonephritis

In partial fulfillment of RLE

Researchers:

Valdez, Shiela Mae A.
Alonzo, Krizzel Marie M.
Sevilla, Rica Allona N.
Flores, Ma. Cristina D.
Mangulabnan, Kimberly M.

March 2015

I. INTRODUCTION Glomerulonephritis literally means inflammation of the glomerulus or the clusters of microscopic blood vessels in the kidney that filter wastes and fluids from the blood. It includes a number of disorders that affect the structure and function of the glomerulus without any prominent inflammation. It is therefore also referred to as glomerular disease or glomerulopathy. In glomerulonephritis, various known and unknown causes trigger immune activity against the glomeruli which damages it. It is characterized by body tissue swelling (edema), high blood pressure, decreased protein in the blood (anemia), and the presence of red blood cells (hematuria) or protein (proteinuria) in the urine. (Christian Nordqvist, Medical News Today, 2009)
Collin, Rull, and Huins (2012) articulated that glomerulonephritis results from a variety of immune and inflammatory mechanisms. It is often described as primary, when it arises on its own and originated on the kidney, or secondary, when glomerular involvement is part of a systemic disease like systemic lupus erythematosus (SLE), and polyarteritis nodosa.
Furthermore, glomerulonephritis can be classified as acute or chronic. In acute glomerulonephritis, the condition starts suddenly and the tissue damage progresses rapidly. With chronic glomerulonephritis, the condition develops gradually and damage becomes extensive after months or years. (Chris et.al 2015)
Acute glomerulonephritis most often occurs as a complication of a throat or skin infection with streptococcus, a type of bacteria.

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