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Neural Plasticity

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Neural Plasticity
Team D
PSY/340
June 5, 2016
Taleshia L. Chandler, Ph.D

Neural Plasticity
The current patient, Stephanie, has experienced a stroke, a temporary interruption of normal blood flow to her brain. There are certain functions and limitations of neural plasticity in the patient’s recovery process. Neuroplasticity is defined as the ability of the nervous system to respond to intrinsic or extrinsic stimuli by reorganizing its structure, function, and connections. While almost all survivors of brain damage experience some behavioral recovery, every patient will vary in his or her recovery process. According to Johansson, MD, PHD (2000), there are several mechanisms that are involved in brain plasticity. Specifically, such as in Stephanie’s case, time is of the essence.
Brain damage can be triggered by a few factors. The most frequent type of stroke known to cause brain damage is known as ischemia, which is the aftermath of any type of confliction in an artery in which a blood clot is created. The less usual type is called a hemorrhage, which is the result of a damaged artery. Once a patient just like Stephanie has experienced a stroke, physicians must immediately determine whether the stroke was ischemic or hemorrhagic. Making such determination is complicating by nature and physicians have their clock ticking because time is limited. (Kalat, 2013, Chapter 5). Knowing that a hemorrhagic stroke is less likely than a ischemic one, physicians take a chance and apply a drug known as tissue plasminogen activator (tPA) which helps break down blood clots but is only beneficial to a ischemic stroke not a hemorrhagic and needs to be induced to the patient within 3 hours of the stroke in order to be effective. (Kalat, 2013, 2009, Chapter 5).
Surprisingly, there is another effective technique which involves the use of cannabinoids, a substance found in marijuana plants. Cannabinoids refrains the liberation of glutamate and perform anti-inflammatory outcomes providing a shield against brain damage. While this appears to be an extraordinary solution to preventing brain damage, unfortunately physicians have yet to consider this a priority method. Physicians have performed such practice on very few stroke victims because cannabinoids just like tPA can only be effective if applied within just a few hours of the victims stroke.
When the stroke happened, some of Stephanie neurons in her brain died because of the damage that was done to the blood arteries that was unable to receive any oxygen. Once a cell starts dying, it will release a chemical inside the brain that causes certain areas in the brain to swell and kill other neurons. “By this damage that has happened and the neurons dying in the motor cortex, the motor functions are limited, if there are still any left to operate.” (Landers, 2004, p.2). A few hours after a stroke some of Stephanie’s motor skills may return because some of the neurons are stunned by the brain being swollen.
The brain may still try to communicate with the motor cortex, but the message may not get to the intended part of the body. For example, a stroke victim usually loses control in one arm and hand because the neurons are no longer there. Neuroplasticity is when the brain tries to find new ways to connect to a cell. Because of this, recovery time for motor functions will vary for every individual. Rehabilitation is one way that Stephanie’s neurons can be rejuvenated and begin working correctly again. There is a chance that Stephanie may have limited mobility for weeks or months after the stroke.

Conclusion In conclusion, every stroke victim’s case is different. As we witness in Stephanie's case, it was crucial to find a solution in order to avoid permanent brain damage. In addition, not only finding which type of stroke Stephanie had was extremely important, but also the time of the stroke. By knowing the time when the stroke happened they could use an immediate solution to prevent brain damage by applying tPA or Cannabinoids. Moreover, in the use of Neuroplasticity, Stephanie has a chance to recover when her brain begins to try and find new ways to connect to a cell. Stephanie's neurons can possibly work again in the near future.

References
Johansson, MD, PHD, B. B. (2000). Brain Plasticity and Stroke Rehabilitation. American Heart Association, 31(), 223-230.
Landers, M. (2004, March). Treatment-induced neuroplasticity following focal injury to the motor cortex. International Journal of Rehabilitation Research, 27 (1), 1-5. http://ovidsp.tx.ovid.com.contentproxy.phoenix.edu. University of Phoenix. (2012 American Heart Association, Inc.). Modulation of Neural Plasticity as a Basis for Stroke Rehabilitation. Retrieved from University of Phoenix, PSY/340 website.

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