...Head to Toe Assessment DO IT! SAY IT! Gather Equipment Enter Room, wash hands 1. Good morning, my name is “Student” & I’m am going to be your student nurse today. How are you doing? 2. Pull Curtain closed * Can you tell me your name and date of birth (match it to the wrist band) * I have reviewed your health history and I see you have already changed to a gown, have you been able to provide a urine specimen? * I am going to do a head to toe assessment on you, this will assess your body systems and their functions During this assessment I am going to ask you some questions, inspect, touch, and listen with my stethoscope to different areas of your body. If you have any questions now or throughout the assessment, please feel free to ask questions and if at any time I make you feel uncomfortable, please let me know. Do you have any questions before we begin? Level of consciousness * Can you tell me where you are and why you are here? * Can you tell me what day it is? **Patient is alert & oriented x 4 to self, place, time, & situation Observe posture, body movements NV and V speech and expressions, mood, expressions **Body posture, speech, NV and verbal communication is appropriate for the situation. Based on conversation mood, feelings and expressions/perceptions are appropriate for this patient. Patient understands current situation at hand...
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...You examine his fingernails and find one that has tiny petechial hemorrhages under it. 1. Name the most likely causative agent of this man's signs and symptoms. 2. Name the most likely infection this man has. 3. What type of culture would a physician most likely order, and why? 4. What is the treatment? 5. What is rheumatic fever? What causes it. Can it be prevented? 1) Streptococcus viridans transmitted through the wound to the bloodstream as a result of the tooth extraction. 2) subacute bacterial endocarditis (endocarditis- inflammation of the endocardium) 3) a blood culture looking for alpha hemolysis because Streptococcus viridans is alpha hemolytic and this would detect the presence of the bacteria in the blood. 5) Rheumatic fever was the cause of the man's heart murmur because the endocardium (lining of heart muscle that covers heart valve) was damaged. A damaged heart valve makes a person more susceptible to bacterial endocarditis because the bacteria easily lodges onto the heart valve. http://jan.ucc.nau.edu/~fpm/bio205/sp-05/cardiovascsyst3=1-8.pdf http://en.wikipedia.org/wiki/Rheumatic_fever...
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...surgery before age 2. Sometimes surgery for a VSD is done during adolescence. Children who have surgery for a VSD may need to take antibiotic medicine for 6 months. This is to protect against an infection of the inner surface of the heart (infective endocarditis). HOME CARE INSTRUCTIONS Medicines • Give your child over-the-counter and prescription medicines only as told by the health care provider. • If your child was prescribed an antibiotic medicine, give it to him or her as told by the health care provider. Do not stop giving the antibiotic even if your child starts to feel better. Dental care • Make sure your child gets regular dental care and brushes and flosses regularly. This will help reduce the risk for infective endocarditis. • Some children with VSDs or repaired VSDs need to take antibiotics before having dental work or other surgical procedures. These medicines help prevent infective endocarditis. Be sure to tell your child's dentist if your child: • Has a VSD. • Has a repaired VSD. • Has had infective endocarditis in the past. • Has an artificial (prosthetic) heart valve. General instructions • Have your child avoid body piercings. Piercings increase the chance that bacteria can get into the body and cause infective endocarditis. If your child has a heart defect and wants a piercing, talk to your child's health care provider first. • If your child has trouble gaining weight, ask the health care provider if your child needs calorie-boosting supplements. • Your...
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...Risk than Mechanical Heart Valves in the Development of Infective Endocarditis? Prosthetic heart valves are, undoubtedly, one of the most profound innovations to be introduced into the medical community. In 1961 the Starr-Edwards ball and cage mitral valve became the first commercially available mechanical heart valve. In 1969, the Bjork-Shiley tilting disk valve emerged, which was then followed by the St. Jude Medical bi-leaflet valve in 1977. In addition to the creation of mechanical valves, bioprosthetic valves were also being developed; and in1968 the Hancock and Carpentier-Edwards porcine mitral valve became a popular alternative to the mechanical valve (Wheatley and Will, 2005, p.1). Over the...
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...Acceptance among and impact on dental practitioners and patients of American Heart Association recommendations for antibiotic prophylaxis * Peter B. Lockhart, DDS1, , , * Nicholas B. Hanson, MPH2, * Helen Ristic, PhD3, * Adriana R. Menezes4, * Larry Baddour, MD5 Available online 19 December 2014 Show more Show less Choose an option to locate/access this article: * Check SFX * doi:10.14219/jada.archive.2013.0230 Get rights and content ABSTRACT Background The 2007 American Heart Association (AHA) guidelines for the prevention of infective endocarditis (IE) called for a major reduction in the number of patients recommended for antibiotic prophylaxis (AP) and redefined the dental procedures considered to put these patients at risk of acquiring the infection. The purpose of the authors' study was to determine the acceptance of these changes among and the impact of the changes on dentists and their patients. Methods The authors sent a survey to a random sample of 5,500 dentists in the United States. Results Ninety-five percent of the 878 respondents indicated that they saw patients who receive AP. More than 75 percent were either satisfied or very satisfied with the AHA guidelines, and the respondents indicated that they believed almost three-quarters of their patients also were pleased. Seventy percent of dentists, however, had patients who took antibiotics before a dental procedure even though the guidelines no longer recommend it. ...
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...Medications 1. Protonix (pantoprazole) – antiulcer agent (proton-pump inhibitors) a. Indications – Erosive esophagitis associated with GERD. Decrease relapse rates of daytime and nighttime heartburn symptoms on patients with GERD. Pathologic gastric hypersecretory conditions. b. Action – Binds to an enzyme in the presence of acidic gastric pH, preventing the final transport of hydrogen ions into the gastric lumen. Diminished accumulation of acid in the gastric lumen, with lessened acid reflux. Healing of duodenal ulcers and esophagitis. Decreased acid secretion in hypersecretory conditions. Pg. 990 2. SynTHROID (levothyroxine) – hormones (thyroid preparations) a. Indications – Thyroid supplementation in hypothyroidism. Treatment or suppression of euthyroid goiters and thryroid cancer. b. Action – Replacement of or supplementation to endogenous thyroid hormones. Principal effect is increasing metabolic rate of body tissues: Promote gluconeogenesis, increase utilization and mobilization of glycogen stores, stimulate protein synthesis, promote cell growth and differentiation, aid in the development of the brain and CNS. Contain T3 (triiodothyronine) and T4 (thyroxine) activity. Replacement in hypothyroidism to restore normal hormonal balance. Suppression of thyroid cancers. Pg. 1219 3. Dulcosate Sodium (colace) – laxative (stool softener) a. Indications – prevention of constipation (in patients who should avoid straining...
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...Arrhythmias * Are common and often benign, but can indicate underlying heart disease. They often occur intermittently and so can be difficult to diagnose. * Can present with palpitations, chest pain, presyncope/syncope, hypotension, pulmonary oedema. Some are asymptomatic e.g. AF. * History taking make sure include: * Precipitating factors, onset, nature (fast/slow, regular/irregular), duration, associated symptoms (chest pain, dyspnoea, collapse). * Causes: * CARDIAC: * MI. * CAD. * LV aneurysm. * Mitral valve disease. * Cardiomyopathy. * Pericarditis. * Myocarditis. * Aberrant conduction pathways. * NON-CARDIAC: * Caffeine. * Smoking. * Alcohol. * Pneumonia. * Drugs (β2 agonist, digoxin, L-dopa, tricyclics). * Metabolic imbalance (K, Ca, Mg, hypoxia, hypercapnia, acidosis, thyroid disease, phaeochromocytoma). * Tests: * Bloods: FBC, U&E, glucose, Ca, Mg, TSH. * ECG: look for signs of IHD, AF, short P-R interval, long QT interval, U waves. * If ECG normal consider doing 24 hour tape. * Echo: look for structural heart disease. * Others: exercise ECG, cardiac catheterisation etc. * Types of Arrhythmias: * Bradycardias: * If asymptomatic and rate >40bpm then no treatment. * Look for cause and stop any drugs that may be the...
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...Please see the note from April 20th for complete details. She was thought to have a sinus infection and was treated with antibiotics. She continued to not feel well and went to the emergency room on April 25th. She says that she woke up with some spots on her fingers and toes, as well as some numbness in her fingers and toes. She had a blood work done at that time, was noted to have a slightly elevated white blood cell count at 13.4, was sent home with recommendations to follow up if she was not feeling better. She was subsequently called the following day because her blood cultures returned positive. She was admitted to the hospital. She was ultimately diagnosed with Staphylococcus lugdunensis bacteremia with a mitral valve infective endocarditis. She was treated in the hospital proper and subsequently, the ICU and ultimately underwent a mitral valve replacement surgery. Just prior to that surgery, it was noted that the amount of [____] seen had quickly increased. She was found to have some visual defect lesions that happened while she was in the hospital and she ultimately underwent an MRI, which showed an embolic lesion to the brain. The lesions on her fingers and toes were thought to be secondary to an embolic events as well. She was hospitalized from April 26th until May 13th. It was suspected that the bacteremia actually did not come from the sinus infection, but from a skin infection. The patient says there are a couple of ways that could happen. One is when...
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...HEART RHYTHMS * 0.4 sec is the PR wave (it is actually the P-Q wave but called the PR wave) and it should be ≤ 0.20 sec. If it’s ≥ than that it indicates 1° heart block. * NSR: normal sinus rhythm, contraction originates from SA node and beats at 60-100 bpm * Sinus (atrial) Bradycardia: SA node discharges at < 60 bpm. TREATMENT is atropine and pacemaker if they become symptomatic. Usually the contractions are irregular but the same distance apart so they are irregular-regular * Sinus Tachycardia (atrial dysrhythmias): SA node discharges at > 100 bpm. Regular but fast; they won’t have heart block because the SA node is firing too rapidly. TREATMENT is BB or CCB to ↓ HR and BP * PAC (Premature Atrial Contraction): impulse travels across atria via abnormal pathway, creating a disturbed P wave. Contraction originates from ectopic focus in atrium other than the SA node. Caffeine and diet pills predispose people to these but they don’t adversely affect health. TREATMENT is none. * Atrial Flutter: atrial tachycardia resulting in recurring, regular sawtooth flutter waves. The ratio of atrial to ventricle contractions is 3:1. TREATMENT is synchronized cardioversion (like defibrillation but the less Joules, 150-200 vs 300, and you must push the “sync” button to synchronize the energy so as to not direct it onto the T wave and send the patient in V-fib) and ablations. * Valve Replacement: patient must be put on blood thinner afterwards and must be anticoagulated...
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...Infectious Diseases Conditions Plague Cause: Yersina pestis ------------------------------------------------- Treatment: Streptomycin (aminoglycoside) and Ciprofloxacin (Fluoroquinolone) ------------------------------------------------- CNS INFECTIONS Meningitis New born cause: Group B streptococcus, Gram-negative enterics, Listeria monocytogenes 2-24 months’ cause: Streptococcus pneumoniae, Neisseria Meningitidis, Haemophilus Influenzae 2-50 years causes: Streptococcus pneumoniae, Neisseria meningitidis 50+ causes: Streptococcus pneumoniae, Neisseria meningitidis, Listeria monocytogenes, Gram-negative enterics Meningococcal Meningitis Cause: Neisseria meningitidis – gram-negative Treatment: * Penicillin/Ampicillin (beta-lactam) * (Chloramphenicol can be substituted in history of penicillin hypersensitivity) * Close contacts – Rifampicin 2 days * Vaccines for prophylaxis – not for serogroup B, sialic acid is identical to the human form Pneumococcal Meningitis Cause: Streptococcus pneumoniae – gram-positive Treatment: * Cefotaxime (3rd generation cephalosporin/beta lactam) – 10-14 days * (If resistance to Cefotaxime – Vancomycin (tricyclic glycopeptide) and Rifampicin) * Adjunctive treatment with Dexamethasone Haemophilus Influenzae Meningitis Cause: Haemophilus influenzae – gram-negative Treatment: * H. influenzae vaccine is available * Cefotaxime (3rd gen cephalosporin/beta lactam) * Adjunctive treatment...
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...Pasteurellaceae, Legionellaceae Sung‐Pin Tseng (曾嵩斌), Ph. D. Department of Medical Laboratory Science and Biotechnology, KMU ext: 2353 E‐mail: tsengsp@kmu.edu.tw Outline (課程要點與學習目標) Characteristic of Pasteurellaceae, Legionellaceae Infections produced by pathogenic species Tests used to identify these species Reference: Chapter 34, 37 Medical Microbiology, 6th Edition Department of Medical Laboratory Science and Biotechnology, KMU Department of Medical Laboratory Science and Biotechnology, KMU Pasteurellaceae (巴斯德桿菌科) Classification – includes four medically important genera Haemophilus Influenzae Misnamed – originally thought to cause the “flu” Haemophilus Pasteurella Aggregatibacter Actinobacillus (rare clinical isolate) Now know that flu is caused by viruses In some cases of flu, H. influenzae is secondary infection Small, G(‐), aerobic or facultative anaerobic rods, requiring enriched media for isolation Department of Medical Laboratory Science and Biotechnology, KMU small, cocobacilli form in sputum from patient with pneumonia thin, pleomorphic form (多形性) from patient with meningitis (80%) Department of Medical Laboratory Science and Biotechnology, KMU Haemophilus Species Haemophilus = “blood loving” Haemophilus spp require hemoglobin for growth: X‐factor ( hemin) Satellite phenomenon H. Influenzae (small colonies surrounding S. aureus colonies) ...
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...weeks - Uncertain diagnosis after one week of study in hospital or - Temp >38.5 - Duration > 2 weeks - Undiagnosed Etiology 1. Noninfectious inflammatory disease - Adult Still’s disease: daily fevers, arthritis, evanescent rash - Giant cell arteritis: headache, loss of vision, symptoms of polymyalgia rheumatica, fever, anemia, high ESR, jaw claudication - Polyarteritis nodosa - Takayasu’s arteritis - Wegener’s - Mixed cryoglobulinemia - Venous thrombosis and thromboembolism - Hematoma - Hyperthyroid, acute thyroiditis - Pheochromocytoma, adrenal insufficiency - Alcoholic hepatitis: fever, hepatomegaly, jaundice, anorexia, 2. Infection - Tuberculosis: most common infection in FUO, PPD positive in 90% of FUO infective endocarditis - Rare infections: leptospirosis, psittacosis, tularemia, melioidosis, secondary syphilis, disseminated gonoccocemia, chronic meningococcemia, visceral leishmaniasis, whipples disease, yersiniosis 3. Malignancy - Lymphoma, especially NHL - Leukemia - Renal cell carcinoma: microscopic hematuria, - Hepatocellular carcinoma - Myelodysplastic syndromes - Multiple myeloma - Atrial myxomas: arthralgia, emboli, hyperglobulinemia 4. Miscellaneous - Drug fever: stimulate an allergic or idiosyncratic reaction, affecting thermoregulation (sulfonamides, penicillins, nitrofurantoin, vancomycin, antimalarials, H1 and H2 blockers, barbiturates, phenytoin, iodides, NSAIDs and salicylates, hydralazine, methyldopa, quinidine, procainamide...
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...Pulmonary / Respiratory Diseases Acute Respiratory Tract Infection J22 Acute Tonsillopharyngitis J06.8 Allergic Rhinitis J30.4 ARDS J80 Aspiration Pneumonia J69.0 Asthma Severe J46 Asthma in Acute Exacerbation J44.1 Asthma, Unspecified J45.9 Atelectasis J98.1 Atypical Pneumonia J15.7 Bronchitis J20.9 Bronchiolitis J21.9 Bronchopneumonia J18.0 Bronchoscopy Z41.8 (1-620) BPD P27.1 (Newborn) Croup J05.0 Empyema Thoracis J86.9 Hyperactive Airway Disease J68.3 Laryngitis J04.0 Laryngomalacia J38.7 Laryngoscopy Z41.8 Pharyngitis J02.9 Post Intubation Subglottic Stenosis J34.2 Pneumonectomy Z41.8 Pneumonia, nonspecific J18.9 Pneumopyothorax, unspec J39.9 Pneumothorax J93.8 Primary Respiratory Tuberculosis (PKI) A16.7 PTB A16.2 Rhinitis J00 Sinusitis J32.9 Thoracotomy Z41.8 Tracheostomy Z93.0 Malfunction J95.0 URTI J06.9 Viral Pneumonia J12.9 Metabolic/Nutrition/Endocrine Dehydration E86.9 / Mild E86.0 / Mod E86.1 / Severe E 86.3 DKA E14.1 DM, unspecified E14.9 type1 E10.9 type2 E11.9 G6PD with anemia D55.0 G6PD – E740 Hypocalcemia E83.5, Dietary E58 Hypokalemia E87.6 Hyponatremia E87.1 Hypothyroidism,unspecified E03.9 Kwashior E40 / Marasmus E42 Lactose Intolerance E73.9 Malnutrition E46 Marasmus E42 Severe Stunting E45 Severe Wasting E43 Vit A Def E50.9 Skin Hypersensitivity/Allergy Nonspecific T78.4 Atopic Dermatitis L20.9 Cellulitis >Unspecified L03.9 >Ear H60.1 >Head L03.8 >Neck L03.8 >Hip/Knee/Leg/Thigh/Hand...
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...Fallot, occurs when there is abnormal passage of blood between chambers or vessels toward the right (Chowdhury, 2007). Due to the abnormal blood flow, “the deoxygenated blood that returns from the tissues returns back to the body without getting reoxygenated,” which causes decreased oxygenation of tissues due to the severe alteration in pulmonary blood flow (Chowdhury, 2007). Among the various types of congenital heart defects, Ventricular Septal Defects (31.03%), Atrial Septal Defects (22.9%), and the Tetralogy of Fallot (14.9%) are the three most common types of congenital heart defects. (CHD Facts and Statistics). Manifestations General manifestations of congenital heart defects include fatigue, heart murmur, increased risk of infective endocarditis, congestive heart failure, growth retardation, increased pulse, increased respirations, dyspnea, orthopnea, and increased risk of upper respiratory infections. Symptoms, however, vary between the different types of congenital heart defects. Due to the incorrect flow of oxygenated blood circulation towards the lungs, heart murmurs, arrhythmias, enlargement of the right side of the heart, enlargement of the pulmonary artery, dizziness, fatigue, and exertional dyspnea are often seen in left to right shunts. In right ot left shunts, symptoms include cyanosis, fainting, heart murmurs, fatigue, clubbing of fingernails, dyspnea, failure to thrive, and growth and developmental disorders. (Chowdhury, 2007) Etiology Genetic Factors While...
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...vessels causes angina. Moreover, as there is an increase in ventricular end-diastolic pressure and impeded relaxation in AS, diminished diastolic coronary filling ensues, resulting in reduced coronary supply to the myocardium. This mismatch between oxygen supply and demand causes angina. Syncope is caused by reduced cerebral perfusion due to transient ventricular fibrillation, reduced CO from atrial fibrillation impeding LV filling, or malfunction of the baroreceptor mechanisms and a vasodepressor response. Dyspnea can be caused by pulmonary vascular congestion resulting from a rise in LV diastolic dysfunction and end pressure, or the incapacity to increase CO when there is fixed obstruction. Moreover, gastrointestinal bleeding and infective endocarditis are symptoms. References: 1Vander AJ, Sherman JH, Luciano DS. Human Physiology: The Mechanisms of Body Function. 4th ed. USA: McGraw-Hill; 1985. p. 315-6. 2Bray JJ, Cragg PA, Macknight ADC, Mills RG. Lecture Notes On Human Physiology. 4th ed. Oxford: Blackwell Science; 1999. p. 338 3Zacharias SK, Goldstein JA. Clinical Assessment of the Severity of Aortic Stenosis. In: Abbas, AE. Aortic Stenosis [Internet]. London: Springer London; 2015. [Cited 2017 Apr 4]. p. 21-8. Available from: https://link-springer-com.ezproxy.flinders.edu.au/chapter/10.1007/978-1-4471-5242-2_2/fulltext.html 4Wood FO, Abbas AE. General Considerations and Etiologies of Aortic Stenosis. In: Abbas, AE. Aortic Stenosis [Internet]. London: Springer London; 2015....
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