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Efferent Arterioles

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Blood that was unable to be filtered in the glomerulus, exits via the efferent arteriole. Efferent arterioles are smaller in diameter than afferent arterioles. This allows pressure to be built up in the glomerulus, aiding in filtration (Elling 245). The blood in the efferent arteriole will eventually exit the kidney via the renal vein. The renal vein is responsible for sending filtered blood back in circulation. Before leaving the kidney, the efferent arteriole will release and absorb minerals and electrolytes due to the close proximity to the loop of Henle. This is regulated by hormones, the body’s level of dehydration, and blood osmolality.
After a brief overview of the anatomy and physiology of the kidney, we can go into further detail regarding …show more content…
There are many factors involving the concentration of urine. This could be due to the body’s level of dehydration, the body’s current blood pressure, hormonal regulation of the kidney, or renal and endocrine diseases. “Glomerular filtration initiates urine formation. The plasma is filtered by the glomerular capillaries with most of this fluid reabsorbed into the bloodstream via the colloid osmotic pressure of the plasma” (Elling 245). The osmotic pressure forces unfiltered blood through arterioles in the glomerulus. The glomerular filtration rate (GFR) is described by the rate of filtration in the glomerulus. Tubular reabsorption and tubular secretion are two other processes involved in urine formation. In tubular reabsorption, fluid moves from the renal tubules into the blood. The kidney regulates the amount of water, electrolytes, and glucose that the body needs at this time. Tubular secretion does just the opposite as fluid moves from the blood into the renal tubule. The composition of urine is modified as it moves through the loop of Henle. The descending limb is water permeable. This means that solutes reabsorb into the renal medulla and water will …show more content…
Kidney’s balance the amount of fluid in the body, detect waste in the blood, and know when to release the vitamins, minerals, and hormones you need to stay alive. There are multiple hormones that affect the function of the kidneys. Renin-Angiotensin System is activated when there is a loss of blood volume or drop in blood pressure ("You & Your Hormones", 2017). Renin is formed by the juxtaglomerular apparatus cells and released at this time. Renin is responsible for converting angiotensinogen into angiotensin I. Angiotensin I is converted into angiotensin II in the presence of the angiotensin-converting enzyme. Angiotensin II is an efficient and potent vasoconstrictor. Along with renin, aldosterone is part of the renin-angiotensin system. Aldosterone causes the tubules of the kidneys to retain sodium, chloride, and water. This reduces urine output and forms concentrated urine (Bledsoe, Porter, Cherry 306). The antidiuretic hormone, ADH, is another hormone that regulates kidney function. ADH regulates the permeability of the distal convoluted tubule and the collecting duct. A lack of ADH results in production of a large volume of dilute urine due to the distal convoluted tubule being less permeable to water. Inversely, if ADH is released, the distal convoluted tubule is more permeable to water leading to water retention and production

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